

“In susceptible hair follicles, dihydrotestosterone (DHT) binds to the androgen receptor, and the hormone-receptor complex activates the genes responsible for the gradual transformation of large terminal follicles to miniaturized follicles. The thinning begins between ages 12 and 40 years, the inheritance pattern is polygenic, and the incidence is the same as in men.”

“Androgenetic alopecia (AGA), also known in women as female pattern hair loss, is caused by androgens in genetically susceptible women and men. These results are similar to those reported in an earlier study using the same device in males with alopecia. Subjects … achieved a 37% increase in hair counts as compared to sham treated control patients.
NORWOOD DUAL TECH SCREEN AND KEYBOARD CLEANER SKIN
The demonstrates that that low level laser (light) treatment of the scalp every other day for 16 weeks using the device is a safe and effective treatment for androgenetic alopecia in healthy women … with Fitzpatrick Skin Types I–IV and Ludwig–Savin Baldness Scale I-2–II-2 baldness patterns. (References are listed at the end of this page.) The following excerpts are intended to represent a consensus of available data. This also supports the premise that there is no difference between men and women with respect to the physical function of hair growth. In the clinical trials for LLLT devices reviewed for this paper, the treatment regimen between genders are the same. This is the main reason Capillus LLC sells only to licensed health care providers rather than over the counter – to ensure the device is used by patients diagnosed with androgenetic alopecia and that these patients have medical advice with regard to optimal regimen. Devices cleared by the FDA for low-level laser therapy are intended for promotion of hair growth in males who have Norwood Hamilton Classifications of IIa to V and in females who have Ludwig (Savin) I-4, II-1, II-2, or frontal patterns of hair loss and have Fitzpatrick skin phototypes I to IV. Physicians use a system known as the Norwood-Hamilton Classification (for men) and the Ludwig-Savin Scale (for women) to describe the degree of hair loss. Capillus, LLC, postulates this lack indicates there is no difference with regard to the physical function of hair regrowth, other than the normal differences found in individuals that is to say, treatment regimen is adjusted by physician prescription based on each individual’s needs, not specific to gender.

Shelly Friedman stated that with regard to low-level laser therapy, he prescribes essentially the same treatment regimen for men and women who present with androgenetic alopecia. The only discussions regarding gender are focused on the differences between the patterns of hair loss, and the increased likelihood with women that hair loss is attributable to reasons other than genetics.Īny reference to treatment difference between men and women is limited to the use of drugs and topicals which target specific hormones which do present differently between the genders. men this very lack of such discussion gives credence to the argument that there is no difference. There is also a lack of published data specifically regarding the treatment (or difference in treatment) of androgenetic alopecia in women vs.

Overall thinning is more prevalent in women, and “receding hairline” or “monk’s spot” are more common in men however, for external strategies for regrowth (i.e., LLLT), there are no published differences in industry literature. We could find no scientific paper postulating that there is a difference in the physical function of hair growth for men vs. This investigator could find no published empirical evidence or reference regarding hair regrowth as a gender specific function, other than pattern, i.e., the form in which hair is lost. The final part of this discussion focuses on gender, specifically, the question whether there is a difference between men and women with regard to the physical function of hair regrowth.
